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Search Results for "DOT1L"

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Rabbit anti-DOT1L Antibody
Bethyl Laboratories® Catalog # A300-954A-T A300-954A

Validated

Rabbit anti-DOT1L Antibody

Validation Performed

All Bethyl Laboratories® antibodies are validated to meet our strict performance standards.

Learn more about the validation performed

Documents (3) Citations ()

Target: DOT1L

Reactivity: Human

Applications:

Host: Rabbit

Clonality: Polyclonal

Conjugate:

Purity:

1 Figure

parentCodeBETHYL-A300-954

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Product has been discontinued
Rabbit anti-DOT1L Antibody
Bethyl Laboratories® Catalog # A300-953A A300-953A-T

Validated

Rabbit anti-DOT1L Antibody

Validation Performed

All Bethyl Laboratories® antibodies are validated to meet our strict performance standards.

Learn more about the validation performed

Documents (5) Citations ()

Target: DOT1L

Reactivity: Human

Applications:

Host: Rabbit

Clonality: Polyclonal

Conjugate:

Purity:

3 Figures

parentCodeBETHYL-A300-953

Bulk Request

For ordering information, see our International Distributors

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Product has been discontinued
Rabbit anti-DOT1L IHC Antibody
Bethyl Laboratories® Catalog # IHC-00494 IHC-00494-T

Validated

Rabbit anti-DOT1L IHC Antibody

Validation Performed

All Bethyl Laboratories® antibodies are validated to meet our strict performance standards.

Learn more about the validation performed

Documents (4)

Target: DOT1L

Reactivity: Human

Applications:

Host: Rabbit

Clonality: Polyclonal

Conjugate:

Purity:

1 Figure

parentCodeBETHYL-IHC-00494

Bulk Request

For ordering information, see our International Distributors

Distributors

Product has been discontinued

Combinatorial targeting of a chromatin complex comprising Dot1L, menin and the tyrosine kinase BAZ1B reveals new therapeutic vulnerability of endocrine-therapy resistant breast cancer
Article

Estrogen receptor alpha (ERα) is a nuclear transcription factor responsible for the regulation of many cell cycle, proliferation, and differentiation genes, especially in breast tissue. Dysregulation of ERα contributes to the formation of breast cancer and is a marker used to determine whether the cancer would be responsive to endocrine therapy. However, many ERα+ breast cancers are unresponsive to endocrine therapy or develop resistance to this treatment strategy. The authors in this paper identify novel combinatorial treatment strategies targeting Dot1L, menin, and BAZ1B.

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